- Are antiviral medicines effective treatments?
- What are the stages of virus replication?
- What blocks viral replication?
- How does interferon interfere with viral replication?
- Which molecule is released from a viral infected cell that interferes with viral replication?
- Can virus be killed by inhibiting their protein synthesis?
Are antiviral medicines effective treatments?
Antivirals interfere with an important enzyme of the influenza virus, called neuraminidase.
The drugs keep the virus from escaping from one cell to infect a neighboring cell.
They are most effective if started within a day or two of the onset of symptoms..
What are the stages of virus replication?
Viral replication involves six steps: attachment, penetration, uncoating, replication, assembly, and release. During attachment and penetration, the virus attaches itself to a host cell and injects its genetic material into it.
What blocks viral replication?
Other examples of viral proteins synthesis inhibitors are Fomivirsen and Interferon. Fomivirsen is an oligonucleotide that binds to CMV mRNA and blocks its replication and thus inhibits the synthesis of proteins that are essential for production of infectious CMV.
How does interferon interfere with viral replication?
Interferon is secreted by cells in response to stimulation by a virus or other foreign substance, but it does not directly inhibit the virus’s multiplication. Rather, it stimulates the infected cells and those nearby to produce proteins that prevent the virus from replicating within them.
Which molecule is released from a viral infected cell that interferes with viral replication?
interferonsVirally infected cells produce and release small proteins called interferons, which play a role in immune protection against viruses. Interferons prevent replication of viruses, by directly interfering with their ability to replicate within an infected cell.
Can virus be killed by inhibiting their protein synthesis?
As a general rule, maximal inhibition of host-cell protein synthesis occurs with viruses that eventually kill their host-cell during the later phases of infection, when viral coat protein is being made in large amounts.